Users on an oral steroid cycle may experience a sudden increase in body weight and muscular development, insomnia, irritability, aggressive combative behavior, puffy face, severe acne, bad breath, a yellowing of the eyes and skin, premature hair loss, hyperactivity, and exaggerated mood swings that include uncontrollable outbursts of anger. Males may also experience testicular shrinkage and can develop female breast tissue on the pectoral muscles. Females may also experience a deeper voice and decreased breast size. Continued use can result in life-threatening situations involving the liver, cardiovascular system, and brain.
Most of my recommendations for considering yourself an advanced steroid user are self-explanatory. Basically, my worry here is that no matter how many cycles you´ve done, if you´re losing half of your gains from each cycle, then you have a lot of work to do to figure out what you´re doing wrong after your cycles end. There´s really no way around that fact & if you´re not keeping half of your gains, then something is going wrong when you end your cycles. Before you jump into an advanced cycle, with multiple compounds and drugs, you need to get your post cycle in order. If you´re losing more than half your gains from every cycle… then something isn´t in check. You aren´t an advanced steroid user you´ve just used a lot of them. But, if you keep most of your gains from each cycle, and meet the other 3 criteria for being advanced, then this cycle is for you!
Androgens are responsible for the growth spurt of adolescence and for the eventual termination of linear growth, which is brought about by fusion of the epiphyseal growth centers. In children, exogenous androgens accelerate linear growth rates but may cause a disproportionate advancement in bone maturation. Use over long periods may result in fusion of the epiphyseal growth centers and termination of growth process. Androgens have been reported to stimulate the production of red blood cells by enhancing the production of erythropoeitic stimulating factor. During exogenous administration of androgens,Â endogenous testosterone Â release is inhibited through feedback inhibition of pituitary luteinizing hormone (LH).